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1·The addition of epinephrine to local anesthetic solutions produces vasoconstriction.

局麻药溶液中加入肾上腺素，使血管收缩。

2·Both acute or chronic hypoxia can cause vasoconstriction and vasoconstriction increase.

急性或慢性的低氧都能使肺血管收缩，血流阻力增大。

3·ET might be the important factor to cause vasoconstriction, resulting in the portent symptoms.

ET可能是导致血管收缩，引起先兆症状的重要因子。

4·Results The intrarenal vasoconstriction in different stages of cirrhosis was significantly difference.

结果不同阶段的肝硬化患者肾内血管收缩程度有明显的差异。

5·The contraction calf muscle, urges the vein vasoconstriction, the acceleration vein blood backflow heart.

收缩小腿肌肉，促使静脉血管收缩，加速静脉血液回流心脏。

6·AIM: to investigate the effects of platelet-derived growth factor BB (PDGF-BB) on vasoconstriction of the vascular vessels.

目的：观察血小板衍生生长因子- BB (PDGF - BB)对大鼠主动脉血管环的收缩作用。

7·Somatostatin and analogues such as octreotide and vapreotide also cause splanchnic vasoconstriction at pharmacological doses.

生长抑素及其类似物如奥曲肽和伐普肽等在药理剂量下也可以引起内脏血管收缩。

8·Furthermore, ECs obtained from these vascular malformations can be used as an experimental model to study SAH-induced vasoconstriction.

此外，这些来源于畸形血管的内皮细胞可作为研究蛛网膜下腔出血引发血管收缩的实验模型。

9·However, in hypoxic pulmonary hypertension rats, 5 - HT - induced vasoconstriction is mediated by both the 5 - HT1Band 5 - HT2A receptor.

但是，慢性低氧肺动脉高压大鼠5 - HT诱导的血管收缩反应是通过5 - HT _ (2 A)和5 - HT _ (1 B)受体介导的。

10·Endothelin is one of the most potent vasoconstriction peptides, and it plays important physiological role by interacting with its receptors.

内皮素是目前已知的缩血管活性最强的生物活性肽之一，通过与内皮素受体结合发挥广泛的生物学活性。

血管收缩

1·ET might be the important factor to cause vasoconstriction, resulting in the portent symptoms.

ET可能是导致血管收缩，引起先兆症状的重要因子。

2·Objective To study the effects of different concentration of desflurane on hypoxic pulmonary vasoconstriction (HPV) during one-lung ventilation (OLV).

目的观测地氟醚麻醉深度对单侧肺通气(OLV)时低氧性肺血管收缩(HPV)的影响。

3·When acute anoxia, the skin, the abdominal cavity internal organs nerve is excited, shrinks the blood vessel function to get the advantage, therefore vasoconstriction;

急性缺氧时，皮肤、腹腔内脏交感神经兴奋，缩血管作用占优势，故血管收缩；

4·The basic pathological alteration of pulmonary hypertension involves three dependent elements:pulmonary vasoconstriction, pulmonary vascular remodeling and pulmonary microvascular thrombogenesis.

前言： 肺动脉高压的基本病理改变有三个方面：肺血管收缩、肺血管重构和肺小血管内微血栓形成。

5·The drugs' mechanism of action remains uncertain, but plausible theories include vasoconstriction, modulation of pro-survival signal transduction pathways, and endothelial cell apoptosis.

药物的作用机制尚不清楚，可能的作用包括血管收缩、调节信号传导通路和促进内皮细胞凋亡。