全部

1·There are other types of CML that do not involve the BCR-ABL gene.

相关疾病还有一些不涉及BCR-ABL基因的其它类型的慢性粒细胞白血病。

2·Inhibiting BCR-ABL tyrosine kinases can impede the growing progress of tumors.

抑制BCR - ABL酪氨酸激酶可有效抑制肿瘤生长。

3·This paper introduces the first, second and some new BCR-ABL tyrosine kinases inhibitors.

本文介绍了第一、二代以及新近开发的BCR - ABL酪氨酸激酶抑制剂。

4·Results The higher valuable the equipments are, the more fixed cost they have with lower BCR.

结果：价格高的设备， 固定成本也高，其成本效益比偏低。

5·Results:The expression signals of BCR/ABL fusion gene were dark violet-blue or brown precipitates.

结果 ：BCR/ABL融合基因表达信号为深 紫 蓝色或棕 色沉淀。

6·These implied that the expression of BCR/ABL gene was related to the active proliferation of bone marrow cells.

结合各例骨髓增生活跃程度，表明BCR/ABL基因表达水平可以反映治疗效果。

7·CONCLUSION: PMA and BCR-ABL chimeric gene can influence the expression of EDAG gene in leukemia cell line K562.

结论：诱导剂佛波酯和BCR - ABL嵌合基因可以影响EDAG基因在白血病细胞系k562中的表达。

8·ABL and BCR, which are located on chromosomes 9 and 22, respectively, are normal genes whose function is still unknown.

而ABL和BCR都是正常基因，分别定位于第9和第22号染色体，但它们的功能目前还不清楚。

9·Methods: to read the related literatures then to review and comment the B lymphocyte development and BCR gene rearrangement.

方法：通过相关文献的阅读，综合评述B细胞发育与BCR基因重排。

10·If STI571 inhibits the BCR-ABL kinase so effectively, what is the result of its inhibition of the ABL kinase in normal cells?

如果STI571可高效地抑制BCR - ABL激酶活性，那它对正常细胞的ABL激酶的抑制会产生什么结果呢？