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1·Objective To explore the specific pathogenic role of glycation products in normal kidney.

目的为探讨糖基化产物对正常肾脏的特异性损害作用。

2·Conclusion: Puerariae can significantly inhibit glycation reaction in rats induced by D-galactose.

结论：葛根素对d -半乳糖诱导的大鼠糖基化反应具有明显的抑制作用。

3·The glycation effect caused by glyoxal, methylglyoxal and other saccharide derivatives is toxic to cells.

糖代谢的衍生物甲基乙二醛等可通过糖化作用产生细胞毒性。

4·RESULTS Inactivation of esterase was induced by glycation, carbamylation and steroid in a concentration .

结果糖化、氨甲酰化和类固醇诱导酯酶失活的作用呈浓度依赖性，类固醇失活效应最快。

5·AIM To establish a rapid method for screening advanced glycation end product (AGE) cross links breaker in vitro.

目的建立一种快速、体外筛选晚期糖基化终产物(age)交联结构裂解剂的方法。

6·An elevated level of skin advanced glycation end products (AGE) is a biomarker of diabetes, the authors explain.

作者解释道，升高的皮肤晚期糖基化终末产物(age)是糖尿病的生物标志物。

7·CONCLUSION Aspirin and ibuprofen show partial protection against the glycation induced inactivation of SOD and cat.

结论阿斯匹林和布洛芬可部分保护糖基化诱导CAT和SOD的失活。

8·Hyperglycemia contributes to greater platelet reactivity through direct effects and by promoting glycation of platelet proteins.

高血糖症通过直接影响和促进血小板蛋白的糖化从而增强血小板的活性。

9·The excess in blood glucose reacts with proteins in tissues to form what are known as advanced glycation end products, or AGE's.

过量血糖反应蛋白在组织中，以何种形式被称为晚期糖化终产物，或年龄的。

10·Objective: To study the effects and the mechanism of advanced glycation end products on bovine vascular smooth muscle cell in vitro.

目的：探讨晚期糖化终产物对离体新生牛胸主动脉血管平滑肌细胞的影响及机制。

糖化

1·The glycation effect caused by glyoxal, methylglyoxal and other saccharide derivatives is toxic to cells.

糖代谢的衍生物甲基乙二醛等可通过糖化作用产生细胞毒性。

2·RESULTS Inactivation of esterase was induced by glycation, carbamylation and steroid in a concentration .

结果糖化、氨甲酰化和类固醇诱导酯酶失活的作用呈浓度依赖性，类固醇失活效应最快。

3·The excess in blood glucose reacts with proteins in tissues to form what are known as advanced glycation end products, or AGE's.

过量血糖反应蛋白在组织中，以何种形式被称为晚期糖化终产物，或年龄的。

4·Antibodies that bind specifically to receptor for advanced glycation end products (RAGE) and RAGE-binding fragments thereof are disclosed.

本发明揭示与晚期糖化终产物受体(RAGE)特异性结合的抗体及其RAGE结合片段。